NAFLD and High Fructose Intake. A Review of Literature
- Carvallo Carvallo, Pamela - Carvallo, Eugenia - Barbosa da Silva, Sandra - Mandarim de Lacerda, Carlos Alberto - del Sol, Mariano
- Datos de publicación:
- INTERNATIONAL JOURNAL OF MORPHOLOGY,Vol.35,676-683,2017
- Higado graso no alcoholico - esteatohepatitis no alcoholica - fibrosis hepatica - Fructosa
- Migración Web of Science 
- One of the most commonly used sweeteners is fructose. Fructose is directly metabolized in the liver and can be converted into glucose, later stored as glycogen constituting a source of energy for the hepatocytes. All excess fructose is converted into lipids by exerting a toxic effect on the liver, similar to that produced by excess of alcohol, and can cause nonalcoholic fatty liver (NAFLD). The aim of this review is to gather recent findings regarding the effect of fructose intake at high concentrations and its relationship with NAFLD.